Jennifer K Trittmann

Contact Information

Call us at: (614) 355-6623

Center for Perinatal Research575 Children’s CrossroadResearch Building 3Columbus, OH 43215 (map)

Learn more about Jennifer K Trittmann

Biography

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Academic and Clinical Areas

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

Awards, Honors & Organizations

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

Research

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Education

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Professional Experience

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

Research Funding

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Contact Information

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Contact Information

Call us at: (614) 355-6623

Center for Perinatal Research575 Children’s CrossroadResearch Building 3Columbus, OH 43215 (map)

Learn more about Jennifer K Trittmann

Biography

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Academic and Clinical Areas

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

Awards, Honors & Organizations

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

Research

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Education

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Professional Experience

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

Research Funding

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Contact Information

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Contact Information

Call us at: (614) 355-6623

Center for Perinatal Research575 Children’s CrossroadResearch Building 3Columbus, OH 43215 (map)

Learn more about Jennifer K Trittmann

Contact Information

• Call us at:
• (614) 355-6623
• Center for Perinatal Research575 Children’s CrossroadResearch Building 3Columbus, OH 43215 (map)

Learn more about Jennifer K Trittmann

Biography

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Biography

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Biography

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Jennifer Trittmann, MD, MPH, is Principal Investigator, Center for Perinatal Research, Abigail Wexner Research Institute at Nationwide Children’s Hospital and an Assistant Professor in the Division of Neonatology, Department of Pediatrics at The Ohio State University College of Medicine. Dr. Trittmann’s research focuses on bronchopulmonary dysplasia (BPD)-associated pulmonary hypertension (PH), a life-threatening neonatal disease characterized by progressive pulmonary vascular endothelial dysfunction and smooth muscle cell proliferation. Currently, there are no available predictive biomarkers or curative therapies for BPD-PH. Our labs at the Abigail Wexner Research Institute have discovered genetic and biochemical markers for BPD-PH including: single polymorphisms in arginase-1 (ARG1), dimethylarginine dimethylaminohydrolase-1 (DDAH1), dual specificity phosphatase-1&5 (DUSP1&5), and plasma levels of asymmetric dimethylarginine (ADMA). Our group has developed endothelial DDAH1 conditional knockout mice for studies after intrauterine inflammation (LPS) and post-natal hyperoxia exposure, a disease model for BPD-PH.

Academic and Clinical Areas

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

Academic and Clinical Areas

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

Academic and Clinical Areas

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

Center for Perinatal Research

Principal Investigator

Neonatology

Principal Investigator

Neonatology Fellowship

Faculty

Primary Department

Center for Perinatal Research

• Center for Perinatal Research
• Principal Investigator
• Neonatology
• Principal Investigator
• Neonatology Fellowship
• Faculty
• Primary Department
• Center for Perinatal Research

Awards, Honors & Organizations

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

Awards, Honors & Organizations

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

Awards, Honors & Organizations

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

Member, NIH LRP Ambassador Program, 2017 - Present Member, American Thoracic Society, 2016 - Present Member, Ohio American Academy of Pediatrics, 2016 - Present Member, Nitric Oxide Society, 2016 - Present Member, Society for Pediatric Research, 2016 - Present Member, American Physiological Society, Respiratory Section, 2014 - Present Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present Member, American Academy of Pediatrics, 2005 - Present Member, March of Dimes, 2005 - Present

• Member, NIH LRP Ambassador Program, 2017 - Present
• Member, American Thoracic Society, 2016 - Present
• Member, Ohio American Academy of Pediatrics, 2016 - Present
• Member, Nitric Oxide Society, 2016 - Present
• Member, Society for Pediatric Research, 2016 - Present
• Member, American Physiological Society, Respiratory Section, 2014 - Present
• Fellow, American Academy of Pediatrics, Neonatal-Perinatal Section, 2008 - Present
• Member, American Academy of Pediatrics, 2005 - Present
• Member, March of Dimes, 2005 - Present

Research

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Research

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Research

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH. Publications

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  

                


                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  

                


                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.

                


                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.

                


                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.

                


                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.

                


                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.

                


                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.

                


                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 

                


                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.

                


                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

View More Publications

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

Overall goals of Dr. Trittmann’s research are to 1) develop a clinical lab biomarker for BPD patients at high risk for developing PH, and 2) discover DDAH1 mechanism of action as it relates to BPD-PH pathogenesis, in order to precisely target treatment to improve outcomes for neonatal patients with BPD-PH.

Interests

Bronchopulmonary Dysplasia Endothelial Cell Biology Pulmonary Hypertension Pulmonary Vascular Biology

Lab(s)

Center for Perinatal Research

• Bronchopulmonary Dysplasia

• Endothelial Cell Biology

• Pulmonary Hypertension

• Pulmonary Vascular Biology

• Center for Perinatal Research

                  Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;  
  
  
  
                  Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;  
  
  
  
                  Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.
  
  
  
                  Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.
  
  
  
                  Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.
  
  
  
                  Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.
  
  
  
                  Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.
  
  
  
                  Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.
  
  
  
                  Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4: 
  
  
  
                  Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.
  
  
  
                  Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.
  
  

View More Publications

• Milton AD, Almazroue H, Jin Y, Zender G, Trittmann JK. DDAH1 SNP rs480414 that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia results in lower nitric oxide production in neonatal cord blood-derived lymphoblastoid cell lines. J Neonatal Perinatal Med. 2021 Jun 17;
• Trittmann JK, Jin Y, Liu Y, Nelin LD. Differential effects of the Src family tyrosine kinases yes and fyn on lipopolysaccharide-induced lung injury in mice. Am J Physiol Lung Cell Mol Physiol. 2021 Jun 9;
• Trittmann JK. Keratin 1: A negative regulator of inflammation and potential treatment for pulmonary arterial hypertension. Acta Physiol (Oxf). 2021 Feb; 231: e13594.
• Talavera MM, Jin Y, Zmuda EJ, Frick J, Liu Y, McBride KL, Nelin LD, Trittmann JK. Single nucleotide polymorphisms in the dual specificity phosphatase genes and risk of necrotizing enterocolitis in premature infant. J Neonatal Perinatal Med. 2020; 13: 373-380.
• Chen LL, Zmuda EJ, Talavera MM, Frick J, Brock GN, Liu Y, Klebanoff MA, Trittmann JK. Dual-specificity phosphatase (DUSP) genetic variants predict pulmonary hypertension in patients with bronchopulmonary dysplasia. Pediatr Res. 2020 Jan; 87: 81-87.
• Trittmann JK, Almazroue H, Jin Y, Nelin LD. DDAH1 regulates apoptosis and angiogenesis in human fetal pulmonary microvascular endothelial cells. Physiol Rep. 2019 Jul; 7: e14150.
• Trittmann JK, Bartenschlag A, Zmuda EJ, Frick J, Stewart WCL, Nelin LD. Using clinical and genetic data to predict pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2018 Dec; 107: 2158-2164.
• Trittmann JK, Velten M, Heyob KM, Almazroue H, Jin Y, Nelin LD, Rogers LK. Arginase and a-smooth muscle actin induction after hyperoxic exposure in a mouse model of bronchopulmonary dysplasia. Clin Exp Pharmacol Physiol. 2018 Jun; 45: 556-562.
• Trittmann JK, Jin Y, Chicoine LG, Liu Y, Chen B, Nelin LD. An arginase-1 SNP that protects against the development of pulmonary hypertension in bronchopulmonary dysplasia enhances NO-mediated apoptosis in lymphocytes. Physiol Rep. 2016 Nov; 4:
• Nelin LD, White HA, Jin Y, Trittmann JK, Chen B, Liu Y. The Src family tyrosine kinases src and yes have differential effects on inflammation-induced apoptosis in human pulmonary microvascular endothelial cells. Am J Physiol Lung Cell Mol Physiol. 2016 May 1; 310: L880-8.
• Trittmann JK, Gastier-Foster JM, Zmuda EJ, Frick J, Rogers LK, Vieland VJ, Chicoine LG, Nelin LD. A single nucleotide polymorphism in the dimethylarginine dimethylaminohydrolase gene is associated with lower risk of pulmonary hypertension in bronchopulmonary dysplasia. Acta Paediatr. 2016 Apr; 105: e170-5.

Education

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Education

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Education

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Date of Appointment at Nationwide Children’s Hospital: 09/30/2011

Fellowship

Nationwide Children’s Hospital

Date Completed: 09/28/2011

Graduate School

The Ohio State University

Date Completed: 06/12/2011

Residency

Nationwide Children’s Hospital

Date Completed: 06/30/2008

Medical School

The Ohio State University

Date Completed: 06/12/2005

Professional Experience

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

Professional Experience

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

Professional Experience

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute2011 - Present The Ohio State University, Assistant Professor, College of Medicine, Department of Pediatrics

2011 - Present Nationwide Children’s Hospital, Principal Investigator, Center for Perinatal Research at The Research Institute

Research Funding

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Research Funding

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Research Funding

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

NIH NHLBI 1K08HL129080 Mentored Clinical Scientist Research Career Development Award Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Novel therapeutic targets for bronchopulmonary dysplasia-associated pulmonary hypertension, Principal Investigator

Contact Information

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Contact Information

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Contact Information

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Center for Perinatal Research

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

Call us at: (614) 355-6623

                    575 Children's CrossroadResearch Building 3Columbus, OH 43215 (map)

• Call us at:
• (614) 355-6623
• 575 Children’s CrossroadResearch Building 3Columbus, OH 43215 (map)